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Considering the role of cysteine as a carbon source in a scenario of high concentrations of cysteine, with no limitation in cyst(e)ine uptake, most part of cysteine will enter the degradation route, reducing the need for cysteine synthesis, dependent on the deviation of homocysteine from the one-carbon metabolism (Figure 2)

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While NAC does not treat progesterone hypersensitivity itself, it may help reduce inflammatory burden and oxidative stress associated with immune activation in autoimmune progesterone dermatitis (APD)

Diabetes Obes Metab (2011) 13:42633